Carbon monoxide causes functional oxygen deprivation by competing with oxygen for haemoglobin and forming carboxyhaemoglobin. Its very high binding affinity reduces the number of sites available for oxygen and impairs unloading from the remaining oxyhaemoglobin. The phrase 'displace oxygen' is a physiological shorthand: carbon monoxide does not remove dissolved oxygen directly, but sharply lowers the blood's effective oxygen-carrying and delivery capacity. The distinction is ecologically important because emission control must target either the directly released precursor or the product formed after atmospheric transport and reaction. Exposure depends on concentration, duration, ventilation, weather, and individual susceptibility, so a pollutant's presence alone does not fully predict biological harm. Atmospheric inversions can trap contaminants near the surface, while wind, turbulence, precipitation, and chemical transformation determine their eventual distribution and removal. At organismal level, respiratory surfaces are especially exposed because large volumes of air contact thin, moist epithelia designed for rapid gas exchange. Sound classification links the source, atmospheric fate, exposure route, and mechanism of toxicity rather than relying only on the pollutant's name.
Ref:
Campbell Biology, Urry et al., 12th Ed., Unit 8 Ecology